Joint pain affects millions of people. It can be due to injuries, autoimmune diseases, or simply the wear and tear of time. Yet, its impact is profound. It limits mobility, impairs quality of life, and often leads to dependence on treatment.
In rheumatology, joint pain is not treated merely as a symptom. It is analyzed in depth. The cause, context, and progression define the therapeutic approach. It's not about "prescribing routinely," but about personalizing each intervention.
The role of nonsteroidal anti-inflammatory drugs (NSAIDs)
Nonsteroidal anti-inflammatory drugs (NSAIDs) are one of the most widely used tools for managing joint pain. Their action inhibits COX enzymes, reducing the production of prostaglandins, which cause inflammation and pain. Among the best known are ibuprofen, naproxen and diclofenac.
Their effectiveness is well documented. They relieve both acute and chronic pain. However, they should be used with caution. Gastrointestinal, renal, and cardiovascular side effects are significant. Therefore, their prolonged use should be supervised.
Are all joint pains treated the same?
No. In rheumatology, the cause of the pain determines the course. Rheumatoid arthritis is not the same as osteoarthritis. While the former is autoimmune, the latter is degenerative. The treatments, although similar in some cases, differ in their objectives.
For rheumatoid arthritis, for example, NSAIDs only control symptoms. They don't modify the disease. This is where DMARDs (disease-modifying antirheumatic drugs), such as methotrexate, come in, which slow joint damage. In contrast, for osteoarthritis, NSAIDs may be sufficient in mild or moderate stages.
Corticosteroids and biologics: other allies in rheumatology
When NSAIDs aren't enough, specialists turn to corticosteroids. These are powerful anti-inflammatories. They're used during flare-ups or critical moments. But they're not recommended long-term due to their adverse effects.
In recent years, biologic therapies have transformed rheumatology. They specifically target the molecules that cause inflammation. TNF, interleukins, and JAK inhibitors are changing the course of diseases such as rheumatoid arthritis and ankylosing spondylitis.
Modern rheumatology: beyond symptomatic treatment
The goal is no longer just to relieve pain. It's to prevent joint damage. Prevent disability. Restore functionality. This requires early diagnosis, close monitoring, and constant adjustments. Today's rheumatology is based on evidence, but also on closeness to the patient.
The approach is comprehensive. It includes physical therapy, weight control, adapted exercise, and psychological support. Medications are important, but not the only ones. Patient education is key. Knowing when to use an anti-inflammatory and when not to can make all the difference.
Self-medication: a common and dangerous risk
Many people take anti-inflammatory drugs on their own. They think that because they're common, they're safe. However, this habit can mask serious illnesses. Furthermore, prolonged unsupervised use can cause ulcers, kidney failure, or high blood pressure.
In rheumatology, we insist on consulting a doctor for any persistent pain. Time is of the essence. The sooner you act, the better the results. Not all pain requires medication. Some should be investigated.
Conclusion: The art of treating pain from the root
Rheumatology does not treat joint pain blindly. It studies it, understands it, and combats it from its source. anti-inflammatories They are useful tools, but not universal. Their use should be guided by a specialist.
Consult the rheumatologist It's not an exaggeration. It's an investment in health, mobility, and quality of life. Because behind the pain, there may be a story worth telling... and treating.
